By Joseph Knoll

The most message of this monograph is that the looks of the mammalian mind being able to gather drives ensured the advance of social existence, and at last ended in the evolution of the human society. This so much refined kind of prepared existence on the earth continues to be within the trial and mistake section of its improvement. It seeks to outgrow the myth-directed period of its historical past and are available to its ultimate nation, the ration-directed human society.

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Extra info for The Brain and Its Self: A Neurochemical Concept of the Innate and Acquired Drives

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2 demonstrates that none of the applied doses of (−)-BPAP was capable of changing the learning performance of rats in the shuttle box. In accord with the findings on cultured rat cortical neurons, the in vivo experiments confirmed that (−)-BPAP, the presently known most potent synthetic mesencephalic enhancer substance, is devoid of a specific enhancer effect on the cortical neurons. The second study of the enhancer effect on cultured telencephalic neurons was performed on cortical cells from 8-day-old chicken embryos (Lohman brown hybrid).

2001). The in vivo dose-dependent enhancer effect of (−)-BPAP is illustrated on noradrenergic (Fig. 7), dopaminergic (Fig. 8), and serotonergic neurons (Fig. 9), respectively. A comparison of the enhancer effect of (−)-BPAP and (−)-deprenyl shows 1. The substantially higher potency of (−)-BPAP than (−)-deprenyl in enhancing the activity of catecholaminergic neurons 2. The characteristic dose-dependency of the enhancer effect of (−)-BPAP on noradrenergic (Fig. 7) and serotonergic neurons (Fig. 9), and 3.

8), and serotonergic neurons (Fig. 9), respectively. A comparison of the enhancer effect of (−)-BPAP and (−)-deprenyl shows 1. The substantially higher potency of (−)-BPAP than (−)-deprenyl in enhancing the activity of catecholaminergic neurons 2. The characteristic dose-dependency of the enhancer effect of (−)-BPAP on noradrenergic (Fig. 7) and serotonergic neurons (Fig. 9), and 3. The highly potent in vivo enhancer effect of (−)-BPAP on serotonergic neurons (Fig. 9) and the lack of this effect on the part of (−)-deprenyl (Fig.

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