By Robert E. Schmidt, Drury R. Reavill, David N. Phalen
Pathology of puppy and Aviary Birds, moment Edition presents a accomplished connection with the gross and histologic gains of ailments obvious in puppy and aviary birds, with greater than 850 photographs depicting disorder lesions.
• Provides an entire source for making a choice on either universal and not-so-common ailments in a variety of avian species
• Includes greater than 850 full-color pictures to teach affliction lesions
• Offers context for the translation of pathologic findings, selling an realizing of the pathogenesis and epizootiology of disease
• Adds details on pigeons and chickens, pathophysiology, analysis and traits, and globally correct diseases
• Aids pathologists, diagnosticians, and avian veterinarians in deciding upon lesions in puppy birds
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Additional info for Pathology of Pet and Aviary Birds
Detail of a pleocellular inflammatory response. 7 Marked swelling and feather loss secondary to sinusitis. inflammation are present and there may be evidence of secondary infection. Finch herpesvirus infection can cause disease throughout the upper respiratory system (see under trachea). ), Mycoplasma, and Chlamydia can cause a sinusitis. Acute sinusitis is characterized by a serous discharge, with swelling and redness in the orbital area and nares (Fig. 7). Histologically there is a thickening of the nasal mucosa or sinus membranes due to infiltrating heterophils, lymphocytes, and plasma cells, as well as hemorrhage, fibrin deposition, and tissue edema (Figs.
29). As the fungal disease progresses, a mixed-cell to histiocytic inflammatory response develops. Histologically in the trachea and bronchi, the fungal mycelia penetrate the walls and combine with inflammatory cells to form caseous, granulomatous nodules (Fig. 30). The syrinx is a common site of a primary focal fungal infection. Many pet birds will have a history of unsupplemented allseed diet suggesting that hypovitaminosis A is a predisposing factor to the development of the tracheal lesions.
In some cases, early diagnosis can keep the infection localized; however, there may be extensive tissue damage that like any process involving the nasal sinus can result in deformities of the beak (Fig. 11). On exfoliative cytology, the organisms are narrow-based budding round yeast with a thick capsule. India-ink stains demonstrate the large heteropolysaccharide capsule. Histologically the numerous yeasts appear as “soap bubbles” separating the tissue’s elements. They have infrequent narrow-based budding and are 5–15 mm in diameter.