By Timothy H Ogilvie

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A) In the early condition, abomasal outflow of HCI is compromised, and reflux occurs into the rumen. The rumen effectively traps H + and CI- ions. K + ions are lost in the urine in deference to H+-ion retention, creating in total a hypochloremic, hypokalemic, metabolic alkalosis. (b) In the advanced condition, a metabolic acidosis may be found related to the state of shock of the animal. (6) Occasionally, the omasum is involved in the abomasal volvulus (omasal-abomasal volvulus), which increases the gravity of the prognosis.

The owner usually notices only the primary disease process (a septic process such as acute mastitis, or an abomasal condition such as LDA or RDA). In calves, the history may be one of poor growth and appetite disturbances. b. Clinical findings (1) There may be no clinical signs of abomasal erosions or ulcers. The lesions may only be evident at slaughter of a normal animal or necropsy of an animal dying from a primary disease process. (2) Alternatively, the clinical signs of a primary disease may be accompanied by signs of abomasal erosion or ulceration.

C. , lymphosarcoma of the abomasum) and abomasal torsion. (2) If adhesive lesions around the reticulum either interfere with vagal nerve function or the mechanics of reticular movement, then reticulorurriinal cyclical activity and eructation waves are compromised, resulting in ruminal gas retention or free gas bloat. In this case, there are no other abnormalities, and the animal will improve transiently if the gas is removed via orogastric tube and recover fully if a rumen fistula is installed.

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